Focus
Selank
Anxiolytic heptapeptide analog of tuftsin studied for anxiety reduction, cognitive enhancement, and stress resilience.
Medical Disclaimer: These products are not intended to diagnose, treat, cure, or prevent any disease. Consult your physician or qualified healthcare provider before starting any new supplement or wellness protocol.
Purchase option
Selank is a synthetic heptapeptide (Thr-Lys-Pro-Arg-Pro-Gly-Pro) developed by the Institute of Molecular Genetics of the Russian Academy of Sciences as a stable analog of the immunomodulatory tetrapeptide tuftsin. It acts on GABA-A receptors and the serotonin system, producing anxiolytic effects without sedation or dependence in animal models. Selank has also been studied for its ability to upregulate BDNF expression, modulate IL-6 and enkephalin metabolism, and improve cognitive function under stress conditions. Each vial contains 5mg at ≥99% purity.
What it does
- Stable heptapeptide analog of tuftsin
- 5mg lyophilized powder — ≥99% purity verified
- Studied for GABA-A modulation and anxiolytic activity without sedation
- BDNF upregulation and cognitive enhancement research tool
- Third-party tested by ISO-certified laboratory
- Cold-chain shipped with Certificate of Analysis
How to use
Reconstitute with 1–2mL bacteriostatic water. Swirl gently until fully dissolved.
Store lyophilized at 2–8°C. Once reconstituted, refrigerate and use within 30 days.
For research use only. Not intended for human consumption.
What’s in the box
- 1 lyophilized vial
- Certificate of Analysis
- Desiccant pack
Product FAQ
Selank modulates GABA-A receptors but lacks the sedative and dependency profile of benzodiazepines in research models. It is studied as a non-sedating anxiolytic with cognitive benefits.
Selank is built on the tuftsin backbone (Thr-Lys-Pro-Arg) with a Pro-Gly-Pro extension that dramatically increases metabolic stability, extending its research utility window compared to tuftsin.
Many researchers pair Selank with DSIP for overlapping anxiety and sleep research. Both target stress-related neuromodulatory pathways via distinct mechanisms.